Research has shown that Alzheimer’s disease patients’ brains contain a large volume of sticky senile plaques. The plaques’ main ingredient is beta-amyloid, a fragment of a normal body protein that goes awry in people with Alzheimer’s disease. beta-amyloid is generated from APP (amyloid precursor protein) through cleaving action of beta and gamma secretase. Some patients produce too much beta-amyloid and others simply do not clear it out of the brain properly. beta-amyloid buildup leads to brain cell death that eventually results in the onset of Alzheimer’s disease. Thus it is demonstrated that protecting the neuronal cells from beta-amyloid insult could prevent the onset of Alzheimer’s disease.